Plastics in Practice (Resident Review) – Details, episodes & analysis

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Plastics in Practice (Resident Review)

Plastics in Practice

Health & Fitness

Frequency: 1 episode/5d. Total Eps: 56

A podcast built for plastic surgery trainees. Each episode reviews CME articles and topics from the ASPS Resident Curriculum, breaking them down into core concepts, clinical pearls, and exam-ready takeaways. Listen on your commute, between cases, or while studying—anywhere you want high-yield plastic surgery learning on the go.

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Hand Infections: Diagnosis and Management

Sunday, May 31, 2026 • Duration 23:55

Hand infections deteriorate fast — the same anatomy that gives the hand its function turns ordinary swelling into ischemia, tendon necrosis, and permanent loss of motion. This episode walks through how to recognize and manage everything from paronychia and felon to pyogenic flexor tenosynovitis, deep space infections, bite injuries, and necrotizing fasciitis — with the surgical pitfalls every resident needs to know cold.

In this episode of Plastics in Practice, we cover the anatomy that makes the hand vulnerable, when to splint and when to cut, the Kanavel signs you'll be quizzed on, the herpetic whitlow trap, the high-mortality red flags of nec fasc, and how rising community-acquired MRSA changes empiric antibiotic selection.

Key takeaways:

• Drainage is non-negotiable for any abscess. Antibiotics are an adjunct, not a substitute.

• Never infiltrate local anesthesia into cellulitis — it spreads the infection. Use regional blocks for deep space infections.

• Herpetic whitlow is medical, not surgical. Cutting risks systemic viral spread. Confirm with Tzanck smear.

• Kanavel's four signs of pyogenic flexor tenosynovitis: semi-flexed digit, fusiform swelling, tenderness along the sheath, pain on passive extension.

• Clenched-fist bite injuries seed the MCP joint. Polymicrobial with Eikenella — admit, x-ray, tetanus, and explore in the OR if the extensor mechanism is breached.

• Necrotizing fasciitis carries up to 40% mortality. Bright shiny skin, nonpitting edema, violaceous patches — broad-spectrum antibiotics and aggressive debridement now.

• Community-acquired MRSA is rising. Culture before empiric antibiotics whenever possible.

This content is for educational purposes only and is not medical advice.

🎧 Full episodes available now:

Instagram: https://www.instagram.com/plasticsinpractice/

Spotify: https://open.spotify.com/show/4Ct8jOgYXP9QJin7QOuG3Z?si=JNcBxQmwT2mfz1LSJZEFKA

Apple: https://podcasts.apple.com/us/podcast/plastics-in-practice-resident-review/id1835564216

YouTube: https://youtube.com/@plasticsinpractice?si=tqLInp5vvsJFKlRO

Amazon: https://music.amazon.com/podcasts/8bef056e-7c87-4224-978e-7e691b04554a/

📘 Free Study Guides: → https://drive.google.com/drive/u/0/folders/12BUldPbCmihG-ndZh6992WqhRYyxw8ZZ

#PlasticSurgery #Residency #HandSurgery #HandInfections #UpperExtremity #SurgicalEducation #PlasticsInPractice #Tenosynovitis

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Upper Extremity Anesthesia Essentials

Thursday, May 28, 2026 • Duration 17:05

Upper extremity surgery is, at its core, a regional anesthesia specialty. Choose the right block in the right place and most hand and arm cases never need general anesthesia.

In this episode of Plastics in Practice, we work through the practical anesthesia decision tree for the upper extremity: brachial plexus block locations and their trade-offs, peripheral nerve blocks at the elbow and wrist, the Bier block, digital block techniques, and the modern, evidence-based truth about epinephrine in the finger.

Key takeaways:

- Severe systemic toxicity is almost always an intravascular accident - aspirate before every injection, and never trust pain as a safety signal in a sedated or proximally blocked patient.

- Interscalene for the shoulder, supraclavicular for fast complete arm anesthesia, infraclavicular for elbow and distal, axillary for the hand.

- Brachial plexus blocks take 15-25 minutes to mature - perform them in a dedicated block area about 30 minutes before incision.

- Bier blocks fit cases under 60 minutes. Keep the tourniquet up for 30 minutes minimum and cycle it down in three releases.

- Epinephrine 1:200,000 is safe in fingers and hands - the necrosis fears come from pre-1950s procaine and cocaine reports.

- Single subcutaneous volar digital block beats the transthecal block on onset and pain.

This content is for educational purposes only and is not medical advice.

Full episodes available now:

Instagram: https://www.instagram.com/plasticsinpractice/

Spotify: https://open.spotify.com/show/4Ct8jOgYXP9QJin7QOuG3Z

Apple: https://podcasts.apple.com/us/podcast/plastics-in-practice-resident-review/id1835564216

Functional Anatomy and Principles of Upper Extremity Surgery

Monday, May 25, 2026 • Duration 17:27

Every hand case rests on the same foundation: knowing which joints need mobility, which need stability, and how to run an acute hand with a trauma-first mindset. Get this layer right, and every chapter after it makes sense.

In this episode of Plastics in Practice, we cover the foundational anatomy and operative principles of the upper extremity: how the thumb, finger, and wrist joints differ in their tolerance for stiffness and instability; the classic deformities (swan-neck, boutonniere, DISI, VISI) and the ligaments that produce them; the acute hand workup from "life over limb" through ER reduction; how to image and describe fractures in anatomic terms rather than eponyms; intrinsic plus splinting; safe tourniquet use; Bruner incisions; and why elevation is the single most important postoperative instruction you'll ever give.

Key takeaways:

MCP joints have low tolerance for stiffness; the DIP tolerates it well - stability matters more there than motion.
Volar plate disruption at the PIP causes swan-neck; central slip rupture causes boutonniere - opposite mechanisms, opposite deformities.
Scapholunate ligament tear leads to DISI; lunotriquetral tear leads to VISI. The lunate follows the ligament that's still intact.
Tourniquet time should be 90 to 120 minutes max to avoid ischemic reperfusion injury; pressure about 100 mmHg above systolic.
Splint in the intrinsic plus position: wrist in slight extension, MCPs flexed, IPs extended.
Routine soft-tissue cases under 2 hours: postop antibiotics show no clear benefit. Open fractures and bony work: 24 hours of perioperative IV coverage.
Bruner zigzag incisions across the volar finger and palm prevent contracture; never cross flexion creases perpendicularly.

This content is for educational purposes only and is not medical advice.

Full episodes available now:

Instagram: https://www.instagram.com/plasticsinpractice/

Lymphedema: Diagnosis and Treatment

Sunday, May 24, 2026 • Duration 23:48

Lymphedema is the chronic disease plastic surgeons are best positioned to treat — and the most commonly mismanaged. After axillary node dissection, up to 50% of breast cancer patients develop it, and most never get the early, disciplined care that prevents progression to fibrofatty disease.

In this episode of Plastics in Practice, we review the essentials of lymphedema management — from how the lymphatic system fails to the diagnostic threshold (2 cm or 200 ml difference), staging by the International Society of Lymphology, the role of Complex Decongestive Therapy as the gold-standard nonsurgical approach, and how to think about surgery: physiologic methods like lymphaticovenous bypass and vascularized lymph node transfer, versus reductive options like liposuction and direct excision. We also cover the late complications residents should not miss — recurrent cellulitis, and the rare but devastating Stewart-Treves lymphangiosarcoma.

Key Takeaways:

Diagnostic threshold: limb circumference difference > 2 cm or volume increase > 200 ml is clinically significant.
Up to 50% of patients with axillary lymph node dissection develop lymphedema; only 4–7% after sentinel node biopsy.
Complex Decongestive Therapy (CDT) is the gold-standard nonsurgical management — Phase 1 intensive (4–6 weeks), Phase 2 maintenance for life.
ISL staging: Stage 0 latent → Stage I pitting → Stage II non-pitting fibrofatty → Stage III lymphostatic elephantiasis.
Physiologic surgery (lymphaticovenous bypass, vascularized lymph node transfer) can reduce limb volume ~35% at 12 months in selected patients.
Liposuction is the workhorse for non-pitting, fibrofatty lymphedema — requires ≥ 600 ml volume difference, failed 3 months of CDT, and lifelong compression after surgery.
Stewart-Treves lymphangiosarcoma: rare but aggressive — 5-year survival < 10%, average survival 19 months after diagnosis. Always biopsy suspicious nodules in a long-standing lymphedematous limb.

This content is for educational purposes only and is not medical advice.

Pressure Sore Fundamentals

Tuesday, May 19, 2026 • Duration 24:18

Pressure sores are the wound the plastic surgeon gets consulted on after everything else has gone wrong — and the recurrence rate is brutal if you skip the basics. The lesson nobody teaches you in residency is that the surgery is the easy part.

In this episode of Plastics in Practice, we review the basics of pressure sore management: the pathophysiology of pressure-time injury, the six-stage NPUAP system, what you absolutely have to optimize preoperatively (albumin, A1c, spasm, contractures, osteomyelitis), how to choose between fasciocutaneous and musculocutaneous flaps for ischial, sacral, and trochanteric defects, and why the recurrence numbers stay high no matter what flap you pick.

Key takeaways:

- Tissue injury starts deep — muscle over bone fails first, the skin lesion is just the tip of the iceberg.

- Capillary perfusion fails above ~32 mm Hg; five minutes of off-loading every two hours is enough to prevent breakdown even at high pressures.

- Pressure x time is parabolic: 500 mm Hg for 2 hours or 100 mm Hg for 10 hours both kill muscle. Skin ulcerates last.

- Optimize before you operate: albumin >2.0 g/dL, A1c <6%, control spasm (baclofen, dantrolene, botulinum toxin), treat osteomyelitis surgically — not medically.

- Avoid primary closure and skin grafts — these wounds have a true tissue deficit. Use flaps.

- Fasciocutaneous vs. myocutaneous: in a 94-patient series there was no difference in recurrence, complications, or morbidity — pick the flap that preserves future options.

- Avoid radical ostectomy — total ischiectomy redistributes pressure and creates the next ulcer.

- Recurrence after flap closure runs ~39%; the most vulnerable window is the first 15–22 months.

- Watch for Marjolin ulcer in long-standing wounds — aggressive SCC with metastatic rates over 60%; wide excision is the answer.

Perineal Reconstruction: Principles and Flap Selection

Sunday, May 17, 2026 • Duration 20:48

Few regions punish poor planning like the perineum — the pelvic outlet sits at the crossroads of the urinary, gynecologic, and GI tracts, and the wrong flap choice in an irradiated or contaminated field gets you back in the OR fast.

In this episode of Plastics in Practice, we walk through the core principles of perineal reconstruction: anatomy and vascular supply, the reconstructive ladder, pelvic dead-space management, and the workhorse flaps you actually need to know — VRAM, gracilis, posterior thigh, Singapore, and the greater omentum. We finish with functional restoration in both female and male patients, including the Cordeiro classification for acquired vaginal defects, scrotal reconstruction after Fournier's, and the basics of microsurgical penile replantation.

Key takeaways:

The perineum is a diamond between the pubic symphysis, ischial tuberosities, and coccyx — anterior urogenital triangle and posterior anal triangle.¹
Pelvic dead space after APR or exenteration is dangerous — fill it with a well-vascularized flap to cut abscess, dehiscence, and bowel-obstruction risk.²
VRAM is the workhorse for combined pelvic and perineal defects; it beats thigh flaps on major complications in this setting.³
Gracilis is the workhorse when you don't want a laparotomy — versatile, low donor morbidity, but watch the short pedicle and unreliable skin paddle for large defects.⁴
Singapore flap is the go-to for vaginal reconstruction — sensate, axial, and preserved by the superficial perineal artery.
Use the Cordeiro classification for acquired vaginal defects: IA → Singapore, IB → VRAM, IIA → rolled rectus, IIB → bilateral gracilis.⁵
Scrotal reconstruction after Fournier's: up to 50% primary closure; otherwise meshed STSG with tunica vaginalis intact and spermatic cords sewn together first.⁶
Penile replantation: microsurgical repair within 6 h warm or 16 h cold ischemia — urethra, tunica albuginea, dorsal artery and vein, dorsal nerve, plus suprapubic cystostomy.⁶

This content is for educational purposes only and is not medical advice.

🎧 Full episodes available now:
Instagram: https://www.instagram.com/plasticsinpractice/
Spotify: https://open.spotify.com/show/4Ct8jOgYXP9QJin7QOuG3Z?si=JNcBxQmwT2mfz1LSJZEFKAApple: https://podcasts.apple.com/us/podcast/plastics-in-practice-resident-review/id1835564216YouTube: https://youtube.com/@plasticsinpractice?si=tqLInp5vvsJFKlROAmazon: https://music.amazon.com/podcasts/8bef056e-7c87-4224-978e-7e691b04554a/📘 Free Study Guides: → https://drive.google.com/drive/u/0/folders/12BUldPbCmihG-ndZh6992WqhRYyxw8ZZ

Foot and Ankle Reconstruction: Core Principles

Friday, May 15, 2026 • Duration 21:57

Saving a foot is rarely about the flap. It's about the angiosome you re-perfuse, the millimeters of debridement you take, and whether you lengthen a tight Achilles before you ever think about closure.

In this episode of Plastics in Practice, we cover the principles of foot and ankle reconstruction: the six angiosomes and how they should drive every revascularization, incision, and flap design; the role of the multidisciplinary team in salvaging a limb that traditionally would have been amputated; and the practical algorithm for moving a chronic wound to closure — debridement, NPWT, dermal templates, and the simple techniques that resolve roughly 90% of these wounds without ever needing a microsurgical free flap. We finish with a location-by-location reconstructive playbook from forefoot to hindfoot, including why Achilles tendon lengthening is the single highest-yield biomechanical move in the diabetic forefoot.

Key takeaways:

- The 5-year mortality after major lower-limb amputation in diabetics exceeds 50% — higher than colon or breast cancer.

- The angiosome concept divides the foot into six vascular territories; direct revascularization of the affected angiosome increases healing 50% and decreases major amputation fourfold.

- Biofilm exists in >90% of chronic wounds and penetrates up to 4 mm — debridement, not coverage, is the rate-limiting step.

- Achilles tendon lengthening cuts diabetic forefoot ulcer recurrence in half at 2 years and is the single highest-yield biomechanical intervention.

- Roughly 90% of foot and ankle wounds heal with simple techniques; only ~10% require flap reconstruction.

- For plantar coverage, the medial plantar fasciocutaneous flap remains the workhorse — sensate, durable, glabrous skin with a wide arc of rotation.

- Free flaps to the foot have the highest failure rate of any anatomic location; anastomose outside the zone of injury and use end-to-side to spare a major vessel.

Lower Extremity Reconstruction: Core Principles

Thursday, May 14, 2026 • Duration 25:43

Lower extremity reconstruction is the most unforgiving testing ground in plastic surgery — every decision is graded by whether the patient can bear weight, walk, and protect a sensate foot for the rest of their life.

In this episode of Plastics in Practice, we walk through the core principles of lower extremity salvage: the zone-of-injury concept, when to fix vs. amputate, fracture management, soft-tissue coverage by leg third, and the trade-offs between limb salvage and a well-fit below-knee amputation.

Key takeaways

Salvage is judged against amputation, not “normal.” The goal is a limb more functional than a prosthesis — loss of the tibial nerve and plantar sensibility is a relative contraindication.
Stabilize the skeleton first. Vascular and nerve repairs done before fixation are routinely disrupted during fracture reduction; external fixation is the workhorse for grade IIIB / IIIC injuries.
Early soft-tissue coverage wins. Closure within 72 hours of injury carries the lowest complication rate; delayed closure (1–6 weeks) climbs to ~50%.
Match the flap to the leg third: gastrocnemius proximal, soleus middle, free tissue distal.
Bone gaps have a tiered answer: cancellous graft for short defects, Ilizarov distraction for 4–8 cm gaps, vascularized fibula up to ~24 cm.
VAC therapy buys time, not closure. It improves the bed and reduces flap size, but use beyond 7 days is associated with higher infection and amputation rates in IIIB tibias.
BKA is a reconstructive choice, not a failure. It adds ~25% to the energy cost of ambulation vs. ~65% for AKA; preserve the knee whenever possible, including with a foot-fillet free flap from the amputated part.

This content is for educational purposes only and is not medical advice.

Abdominal Wall Reconstruction: Core Principles

Wednesday, May 13, 2026 • Duration 22:30

Abdominal wall reconstruction is where plastic surgery principles get tested in real time — and it's where most failures start with one wrong assumption about force distribution.

In this episode of Plastics in Practice, we review the basics of abdominal wall reconstruction: how to think about the abdomen as a pressurized cylinder, why supported repairs beat bridged repairs almost every time, how to read the components separation technique like a physiology problem instead of a memorization problem, and how to choose between synthetic and bioprosthetic mesh in a contaminated field. We close with soft tissue strategy — when to skin graft early, when to reach for a perforator flap, and how to decide between rebuilding the wall first or rebuilding the cover first.

Key takeaways:

Hernias expand because sutures cut through tissue like a wire through ice — load-sharing supported repairs are the answer, not bigger primary sutures.
Large hernias convert isometric abdominal contraction into isotonic shortening, derailing diaphragm function and torso mechanics — repair restores physiology, not just anatomy.
Components separation can move each rectus 8–10 cm to the midline through external oblique release alone; preserving periumbilical perforators is what keeps the skin alive.
Synthetic mesh is for clean fields. Bioprosthetic mesh is for contamination, radiation, bowel suture lines, or any field where the soft tissue cover might fail.
In the open abdomen, early skin grafting over granulated bowel is your friend — it heals in two dimensions and tolerates poor nutrition far better than flaps.
If the hernia is expanding, it's ready to repair — that means the adhesions have softened enough to dissect cleanly.

This content is for educational purposes only and is not medical advice.

Chest Wall Reconstruction Essentials

Tuesday, May 12, 2026 • Duration 28:18

Chest wall reconstruction is where rigid skeletal support and versatile soft tissue coverage meet — and where a wrong flap choice or a missed debridement step turns a small defect into a respiratory disaster.

In this episode of Plastics in Practice, we review the basics of chest wall reconstruction: the anatomy that drives every decision, when you actually need to rebuild the skeleton, how to classify and treat sternal wound infections, and how to pick the right flap based on the location and depth of the defect.

Key takeaways:

Skeletal reconstruction is classically indicated for defects involving four or more ribs or greater than 5 cm in diameter — but posterior and superior defects often tolerate soft tissue coverage alone.¹
Methylmethacrylate–mesh "sandwich" gives you rigid protection for large anterior defects, but its rigidity can cost you respiratory comfort.¹
Pectoralis major is the workhorse for the upper and middle sternum; for the lower sternum, think IMA-perforator turnover flap or rectus abdominis (VRAM/TRAM).²
Omentum is your bailout when local flaps fail — large surface area, thin, perfect for wrapping vascular grafts — but you're paying for it with a laparotomy.³
Bilateral IMA harvest strips the sternum of its segmental blood supply and dramatically raises mediastinitis risk; sternal plating is reserved for high-risk re-operative patients.⁴
Sternal wound infections are classified by timing: Class 1 (early, sterile), Class 2 (1–3 weeks, purulent mediastinitis), Class 3 (chronic draining sinus). Class 2 and 3 demand thorough debridement plus flap coverage.⁵
Single-stage radical debridement with concurrent flap coverage has comparable success to staged debridement — pick based on the patient and the wound, not dogma.⁵

This content is for educational purposes only and is not medical advice.

Plastics in Practice (Resident Review) – Details, episodes & analysis

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Plastics in Practice (Resident Review) – Details, episodes & analysis

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